Shared computer activation is required for scenarios where multiple users share the same computer and the users are logging in with their own account. Normally, users can install and activate Microsoft 365 Apps only on a limited number of devices, such as 5 PCs. Using Microsoft 365 Apps with shared computer activation enabled doesn't count against that limit. If your users have dedicated computers and no other users work on those computers, you use product key activation for Microsoft 365 Apps.
To use shared computer activation, you need an Office 365 (or Microsoft 365) plan that includes Microsoft 365 Apps and also supports shared computer activation. Shared computer activation is available for the following plans:
New Configurator 360 2013 activation
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If Microsoft 365 Apps is already installed and you want to enable shared computer activation, there are three options to choose from. A reinstallation isn't required. The device must be rebooted in order to apply the change.
Use Group Policy by downloading the most current Administrative Template files (ADMX/ADML) for Office and enable the "Use shared computer activation" policy. This policy is found under Computer Configuration\Policies\Administrative Templates\Microsoft Office 2016 (Machine)\Licensing Settings.
The Support and Recovery Assistant fully automates all of the steps to verify requirements for shared computer activation and to enable shared computer activation. It's available in two versions: enterprise and UI. Use the version that's best suited for your situation.
There is a known issue where, on devices with both shared computer activation and viewer mode enabled, licensed users are incorrectly placed in viewer mode. This issue is fixed in Version 2205 and later.
Activation limits Normally, users can install and activate Microsoft 365 Apps only on a limited number of devices, such as 5 PCs. Using Microsoft 365 Apps with shared computer activation enabled doesn't count against that limit.
Single sign-on recommended The use of single sign-on (SSO) is recommended to reduce how often users are prompted to sign in for activation. With single sign-on configured, Microsoft 365 Apps is activated using the user credentials that the user provides to sign in to Windows, as long as the user has been assigned a license for Microsoft 365 Apps. For more information, see Microsoft 365 identity models and Azure Active Directory.
If you're using Group Policy, download the most current Administrative Template files (ADMX/ADML) for Office and enable the "Specify the location to save the licensing token used by shared computer activation" policy setting. This policy setting is found under Computer Configuration\Policies\Administrative Templates\Microsoft Office 2016 (Machine)\Licensing Settings.
Microsoft Office includes popular tools like Word, Excel, PowerPoint, Outlook, Access, etc. Depending on how to get or purchase Office, the Office activation process may be different. This post teaches you how to activate Microsoft Office 365/2021/2019/2016/2013 by using different ways. A free Office file recovery tool is also provided to help you recover deleted or lost Office documents with ease.
For schools, companies, or other organizations, you can use a KMS software product to automatically activate Office products without entering a license key. The Key Management Service (KMS) is an activation service that allows organizations to activate systems within their own network. The KMS client can look for a local KMS server and activate the software like Windows OS or Office products for 180 days. After 180 days, you may need to reactivate Office again by using the KMS tool.
When you go through activation of your Office product and it fails, you will see Unlicensed Product or Non-commercial use / Unlicensed Product in the title bar of your Office apps. This will cause most features of Office to be disabled. You will need to fix the problem that is causing the activation to fail.
The Activation Troubleshooter runs on Windows PCs and can fix activation errors for Office Home & Student, Office Home & Business, Office Professional, and individual Office apps, like Visio, Project, Word, and Outlook.
* Your credit monitoring and alerts, Social Security number monitoring, Identity Theft Insurance and Identity Fraud Support Service will begin automatically upon enrollment.** Your other additional benefits will not be available or released to you unless you visit 53identityalert.com and use the member account number to create log in credentials and then access these benefits. Or you may call us at 1-800-972-3030 to request activation materials to be sent to you through the mail.
Based on my searching, there is a blog descripts that some VPN would cause the issue on office 2013, the root cause of the issue seems to be that Office 2013 took a dependency on NLA (Network Location Awareness), and some VPN solutions do not work properly with NLA. More details for your reference: Office 2013 can not check in files when LAN does not have internet access or over some VPN's.
Since many users meet the similar issue, maybe the related activation server was temporarily unavailable, please wait awhile and try again later to sign in and update the license by clicking Check for updated license hyperlink.
10. Domarad, J., Grisak, P., and Bolger, J. (2013), Improving Crosswalk Safety: Rectangular Rapid-Flashing Beacons (RRFB) Trial in Calgary,Canada Institute of Transportation Compendium, available at: conferences/compendium/20 1 3_ Pedestrian_ImprovingCrosswalkSafety. pdf.
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For the mechanism, please refer to the text on1,3-dipolar cycloaddition. This reaction is highly exothermic, but the high activation barrier is responsible for a very low reaction rate, even at elevated temperature. Another drawback is the formation of regioisomers, as the two possible HOMO-LUMO interactions of the substrates are closely related in terms of energy. The thermal reaction therefore often gives approximately 1:1 mixtures of both the 1,4-substituted and the 1,5-substituted regioisomers.
Imran Haq, James A. Irving, Sarah V. Faull, Jennifer A. Dickens, Adriana Ordóñez, Didier Belorgey, Bibek Gooptu, David A. Lomas; Reactive centre loop mutants of α-1-antitrypsin reveal position-specific effects on intermediate formation along the polymerization pathway. Biosci Rep 1 June 2013; 33 (3): e00046. doi:
where k1,fr is a first-order activation rate constant, kapp,fr is the apparent second-order association rate constant, M is a non-activated monomer, Ipol a polymerization intermediate and P the species reported by FRET. Exploration of alternative pathways with greater complexity, including reversible activation and mixed intermediate species, failed to yield improved fits; similarly, removal of the initial unimolecular step resulted in a much poorer correspondence with the data.
As polymerization is the consequence of distinct activation and self-association processes, we sought to determine whether the effects of the mutations could be explained by differences in intermediate formation. It has been observed previously that intermediate formation can be followed spectroscopically by CD, ANS or bis-ANS binding, and by two-phase analysis of change in intrinsic tryptophan fluorescence [14,40].
At 55C, the bis-ANS dye was found to report a phase with significantly faster rates (k1,ans) for all proteins than that reported by the FRET and intrinsic fluorescence assays (Figure 4D, circles). This is consistent with its ability to detect an activation step that precedes polymer formation [40]. Interestingly, however, only P14P12 showed a significant decrease in the rate of activation with respect to the ATC232S control (one-way ANOVA, P
The unimolecular phase of polymerization can be followed using CD spectroscopy at 222 nm, which reports changes in secondary structure content [14]. As the bis-ANS data did not account for differences in the rate of polymerization, and since the use of an ANS-based dye may perturb structural equilibria [19], we used this technique to explore other potential effects on the activation to Ipol.
It has been noted previously that CD of plasma α1-antitrypsin reports a single transition consistent with activation and not polymerization [14]. The behaviour of the control variant is consistent with this. While the CD progress curves of the mutants demonstrated a measurable second phase, the significance of this was not clear, as the rate (k2,cd) did not appear to correlate with other measures of polymerization or activation (Table 3). This transition may therefore represent a secondary structural change not directly associated with the polymerization mechanism.
In contrast, P6P4 does not decrease the rate of early changes reported by bis-ANS or CD, but increases the activation energy required to transit between the intermediate ensemble these methods report, and the terminal self-associated form. As this increase is reflected in the intermolecular rate constant, this is most likely the consequence of interference with an intermolecular interaction. It is nevertheless possible that the mutant interferes with the progression to a late-stage intermediate form; however it is noteworthy that the P14P12 variant shows no effects on the activation energy despite directly affecting the bis-ANS-binding intermediate state, arguing against significant influence of the first-order step on the calculation of the second-order rate constant.
Haq, I. and Irving, J.A. and Faull, S.V. and Dickens, J.A. and Ordóñez, A. and Belorgey, D. and Gooptu, Bibek and Lomas, D.A. (2013)Reactive centre loop mutants of α-1-antitrypsin reveal position-specific effects on intermediate formation along the polymerization pathway. Bioscience Reports 33 (3), pp. 499-511. ISSN 0144-8463. 2ff7e9595c
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